Supply Desonide cas no 638-94-8

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Supply Desonide cas no 638-94-8
product Name Desonide
Molecular Formula C24H32O6
Molecular Weight 416.5073
InChI InChI=1/C24H32O6/c1-21(2)29-19-10-16-15-6-5-13-9-14(26)7-8-22(13,3)20(15)17(27)11-23(16,4)24(19,30-21)18(28)12-25/h7-9,15-17,19-20,25,27H,5-6,10-12H2,1-4H3/t15-,16-,17-,19+,20+,22-,23-,24+/m0/s1
CAS Registry Number 638-94-8
EINECS 211-351-6
Molecular Structure 638-94-8 Desonide

Density 1.3g/cm3
Boiling point 580.1°C at 760 mmHg
Refractive index 1.598
Flash point 196.9°C
Vapour Pressur 7.16E-16mmHg at 25°C

Anti-inflammatory Raw Powder Desonide Without Side Effects CAS 638-94-8


1. Desonide Description:
Name:Desonide
English Synonyms: (11b,16a)-11,21-dihydroxy-16,17-((1-methylethylidene)bis(oxy))-pregna-1,4-diene-3,20-dione;16-alpha-hydroxyprednisole-16,17-acetonide;17-acetonide,16-alpha-hydroxy-prednisolone-1;steroderm
CAS NO.: 638-94-8
EINECS: 211-351-6
Molecular Formula:C24H32O6
Molecular Weight:416.51
Assay: 99%
Chemical Properties Off-White Solid
Usage: With acetal-based adrenal cortex hormones.Anti-inflammatory. Desonide has also been found as an impurity in budesonide (B689490).
Packing:foil bag or as required

2. Desonide Usage:
This medication is used to treat a variety of skin conditions (e.g., eczema, dermatitis,allergies, rash). Desonide reduces the swelling, itching and redness that can occur in these types of conditions. Desonide is a mild corticosteroid.

Desonide Ointment, 0.05% is a low potency corticosteroid indicated for the relief of the inflammatory and pruritic manifestations of corticosteroid responsive dermatoses.
It should not be used for longer than two weeks unless directed by a physician.

3. Desonide Application:
Like other topical corticosteroids, Desonide has anti-inflammatory, antipruritic, and vasoconstrictive properties. The mechanism of the anti-inflammatory activity of the topical steroids, in general, is unclear. However corticosteroids are thought to act by the induction of phospholipase A2 inhibitory proteins, collectively called, lipocortins. It is postulated that these proteins control the biosynthesis of potent mediators of inflammation such as prostaglandins and leukotrienes by inhibiting the release of their common precursor arachidonic acid. Arachidonic acid is released from membrane phospholipids by phospholipase A2.

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