Pharmaceutical Raw Materials Hydrocortisone for Anti-Inflammatory Allergy CAS50-23-7

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Pharmaceutical Raw Materials Hydrocortisone for Anti-Inflammatory Allergy CAS50-23-7 Basic Info. Product Name:Hydrocortisone CAS: 50-23-7 MF: C21H30O5 MW: 362.46 EINECS: 200-020-1 Assay: 98% Appearance: white powder Package: 1kg/foil bag Grade:Medicine grade Description about Hydrocortisone : Suitable for contact dermatitis, lichen planus, etc. It is currently the most important supplementary glucocorticoid drugs, extensive drug effects, versatile, indispensable clinical drugs. Adrenal crisis in the rescue, anaphylactic shock, status asthmaticus critically ill played an important role. In systemic lupus erythematosus, rheumatism, autoimmune vasculitis treatment of autoimmune diseases is an important and indispensable therapy. The advantage of this drug is its ethanol and hydrocortisone sodium succinate intravenously directly and quickly to play a role for the rescue of critically ill patients and shock therapy for certain diseases, generally do not cause peptic ulcers, purpura and spirit symptoms, but the row of potassium and sodium retention is stronger. Short-term large doses of hydrocortisone (24h infusion 2000 ~ 4000mg, once every 2 to 3 days) for some critically ill with the rescue, to receive good results, such as the rescue of septic shock, status asthmaticus , adult respiratory distress syndrome, short-term application, generally no adverse reactions. Application fields: 1. Biochemical studies, adrenal cortical hormone drugs. 2. Glucocorticoids have anti-inflammatory, anti allergic, anti toxin, anti shock four role. 3. Adrenal corticoids Pharmacological action : Anti-inflammatory effects: anti-inflammatory effects of hydrocortisone cortisone 1.25 times, in the pharmacological doses of infectious and non-infectious inflammation are inhibited. Hydrocortisone increases vascular tone, alleviate congestion, reducing capillary permeability, and therefore reduce the exudation, edema; by inhibition of inflammatory cells (including macrophages and leukocytes) aggregation at sites of inflammation, and inhibit phagocytosis cell function, lysosomal membrane stability, prevent complement chemical involved in inflammation and inflammatory mediators (such as prostaglandins, thromboxane, leukotriene) synthesis and release to relieve redness, swelling, heat, pain and other symptoms.

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